If only a few out of millions and even billions of individuals are to survive and reproduce, then there is some difficulty believing that it should really be the fittest who would do so.In addition, he was interviewed two days ago by Paul Nelson, in a podcast posted very recently by the Center for Science and Culture of the Discovery Institute, on their blog Evolution News and Views. You will find it here. He makes the same point (while Nelson misunderstands him and keeps raising an unrelated point about protein spaces). It is a stunning thought that evolutionary biologists have ignored this issue. Have they? Have population geneticists ever thought about this? Well, actually they have, starting nearly 90 years ago. And the calculations that they made do not offer support to Dr. Lönnig. Let me explain ... The standard model In population genetics, the standard model of genetic drift in finite populations is the Wright-Fisher model, introduced in 1930 and in 1932 by those two founders of that discipline. The other great founder, JBS Haldane, used a nearly equivalent model in 1927, though discussed less explicitly. In a Wright-Fisher model there are N parents, each of whom produces a very large but equal number of gametes. So large, that it is assumed that there are an infinite number of them, each parent contributing equally. These gametes then combine at random to form all possible genotypes, each in exactly its expected frequency. If natural selection occurs, it then shifts the genotype frequencies in precisely the expected way. Finally, density-dependent mortality occurs, leaving only N survivors, so that the population size is maintained. It impacts all genotypes equally, so that the N surviving adults are in effect a random sample from the genotypes that survived natural selection. This sampling of adult survivors causes the genetic drift. A numerical example For example, if we have a population of a haploid species with N = 10,000 individuals with two alleles A and a at equal frequencies, each of them will produce a vast number of gametes, equal numbers from the two genotypes. Among the gametes they will be in a 1:1 ratio. Now if the a genotype has viability 1% lower, after that mortality their numbers will stand in the ratio of 1 : 0.99. So after this mortality the frequency of the A genotype is 1/(1+0.99) = 0.50251256. These young individuals then die randomly in freak weather, are eaten randomly by predators, are run over by trucks, and so on. All the haphazard random mortality that Lönnig is worried about. Finally a random 10,000 of them are chosen to win the lottery and survive. As Lönnig says, there are all sorts of outcomes possible. Will the natural selection be effective? Lönnig obviously thinks not. But we can do the calculations. Will the frequency of the A genotype increase? Each of the 10,000 survivors is a randomly drawn offspring, and 0.50251256 of those are A. So it's just like tossing a coin 10,000 times, when the probability of Heads is 0.50251256. The outcome A simple binomial distribution shows that among the adult survivors the probability that the A genotype is more frequent than a is 0.681725. In all the random dying and random survival, the frequency of A rises more often than it falls. Now that is one generation. Further rounds of reproduction and survival, with the same fitnesses, will ultimately lead to the frequency of A either rising to 100% or falling to zero. What is the probability that, starting with equal frequencies, we end up with A winning out? For that we move from Fisher and Wright's models to calculations by PAP Moran (1958) and Motoo Kimura (1962). Let's leave out the details, and open the envelope. And the probability is ... 0.999999... and so on until there are 43 of those 9s. An explanation We can conclude that Dr. Lönnig is not familiar with theoretical population genetics. He is a retired plant breeder at the Max Planck Institute for Plant Breeding Research, who specialized in mutation effects in such plants as the "husk tomato" Physalis pubescens. I can understand why he might not have studied population genetics thoroughly. But why then is he holding forth on the topic? This is easily explained. He is also a creationist, associated with the German creationist organization Wort und Wissen. He formerly posted creationist material on his homepage at his Max Planck Institute. In a controversial move, the Institute forbade him to do this. If Dr. Lönnig wants to understand these matters more, I recommend to him that he visit a gambling casino -- in spite of the wild uncertainty of individual gambles, he might be surprised at how often he would lose his pocket money playing games that are mostly random, but slightly biased in favor of the house.
A devastating critique of population genetics? The Discovery Institute thinks so
A retired European geneticist, Wolf-Ekkehard Lönnig, has made a point that he feels is devastating to population genetic arguments about the effectiveness of natural selection. In a post
at the Discovery Institute's blog Evolution News and Views. He pointed to an argument he made in 2001 in an encyclopedia article. The essence of his criticism is that many organisms produce very large numbers of gametes, or of newborn offspring. Most of those must die. Then
216 Comments
https://me.yahoo.com/a/JxVN0eQFqtmgoY7wC1cZM44ET_iAanxHQmLgYgX_Zhn8#57cad · 4 May 2016
It's like probabilities are magic to them--not understandable, not predictable, yet somehow devastating to evolution.
Imagine, though, if human brains had evolved. Then some people might not really be reliable arbiters of issues like evolution and probability, unlike if God gave people certain knowledge, in at least the crucial matters. Fortunately, they know that they must be right, no matter how confused they may seem about basic issues.
Glen Davidson
https://me.yahoo.com/a/PkZsUe11rOBbyBPTMFoa6pUDJUQQjTxv#0c747 · 5 May 2016
I admit I am a total novice at this, but may I ask the following query:
A population has a certain environmental distribution and produces a number of offspring - each of which finds itself in a different environment - in that environment there is a probability p(E) they will survive.
The population has two allelles A and a. Overall there is a p(A) and p(a) that an individual will survive - lets say p(A) is greater.
But in some environments p(E|a) is higher than p(E|A); in others the opposite is the case - which shakes out to give the overall p(A) and p(a).
So whether the population prospers or declines depends upon two things - its distribution within the environment (if the population has all been washed to a hostile environment its prospects are dim) and the fitness of the genes to survive in that environment.
After a generation there is a new population - again distributed (most likely differently now) within the environment.
My untutored feeling is that the variation in environments will have a very significant impact on whether the species prospers or declines - I'm also fascinated by the assumption of a constant population in the population genetics model - it seems such an unrealistic constraint.
Gene fitness clearly affects survivability, but so too does environmental variation - which of these two factors has a greater effect on the survivability of the entire species seems very complex and not to me obvious.
The distribution of the species may be such that the environmental constraints have a far bigger impact on its survival than genetic ones.
I don't want to give any support to creationists - and I assume far more complex modelling has been done in the last 75 years building on the models of Haldane, Wright and Fisher, but I can't help feeling the OP misses a point
I'd love to understand how Modern evolutionary theory has built on the shoulders of these giants adding environmental issues into their models to help us try to understand when evolution is driven by Natural Selection, when it is driven by genetic drift and when it is driven by environmental factors - remember those poor dinosaurs and the radiations after their removal from the environment.
A model with a static population in a constant environment where population distributions can depend upon tiny variations in genes which even though tiny give statistical guarantees of gene success are all very well, but effective population sizes and environments show these assumptions don't hold very well for the real world - have we built better assumptions into our models since these early models - I'd love to know.
eric · 5 May 2016
DS · 5 May 2016
I guess that no creationist is capable of understanding the concept of "random". They seem to mangle it every time. You would think that someone who studied mutations for a living would at least have a clue. Oh well, yet another creationist spouting off about things he knows nothing about shouldn't do too much harm. After all, he con only convince the willfully ignorant.
Of course, many potentially beneficial mutations are lost due to drift. Of course natural selection still operates. Of course evolution still works. Of course these issues have already =been dealt with mathematically. Of course creationists are completely ignorant of this history. CUltivating and propagating ignorance is apparently their job description. SInce the guy apparently didn't use any math to support his preconceptions, it is obvious that he was just using his incredulity as evidence, same as always. Next he'll be saying that the human eye couldn't evolve!
Joe Felsenstein · 5 May 2016
(I have added subheadings to my post, to break up the big block of text).
Masked Panda c747: I am not sure what you are asking. Your notation is unusual. I normally would use p(A) and p(a) as the frequencies of the two genotypes, not their fitnesses, for example. And my objective here is to analyze Lönnig's assertions that in simple cases genetic drift will eliminate the effect of natural selection, not to ask whether the population survives. (In these models it does survive because there is a large reproductive excess, and a strong density-dependent population size regulation).
eric: Yes, you can analyze cases where environments change, and cases where the population is distributed over environments. Lots of work on this has been done. In fact I wrote a review article in 1976 in Annual Review of Genetics entitles "The theoretical population genetics of variable selection and migration" with 152 references. That was 40 years ago and there have been many more papers on those topics since.
The present simple model, with one population and the same fitnesses every generation is sufficient to show that Lönnig doesn't know the theory relevant to predicting outcomes in even simple cases.
Michael Fugate · 5 May 2016
Joe Felsenstein · 5 May 2016
Daniel · 5 May 2016
It's almost like they don't understand how does a casino make money when they only have a 2% house advantage on their games
Robert Byers · 5 May 2016
If only a few out of millions/billions survive to reproduce it does not make sense that in such a small number the fittest would survive. its too small or rather all the rest didn't survive and so why would these FIT ones survive.
i think he's saying there is no reason these few aree more fit then all those who didn't survive. They couldn't be recognized by nature as more fit. Its a common sense thing against math/probability ideas.
he is a sharp scientist and knows in practical ways about mutations.
Thats where he is coming from. Indeed not the blackboard but real biology. a wee bit of instinct too.
phhht · 5 May 2016
phhht · 5 May 2016
Henry J · 5 May 2016
Yeah, that word "fittest" is a huge oversimplification of what it means in this context.
stevaroni · 5 May 2016
Rolf · 6 May 2016
quentin-long · 6 May 2016
Anton Mates · 6 May 2016
gnome de net · 6 May 2016
TomS · 6 May 2016
Joe Felsenstein · 6 May 2016
https://me.yahoo.com/a/JxVN0eQFqtmgoY7wC1cZM44ET_iAanxHQmLgYgX_Zhn8#57cad · 6 May 2016
Henry J · 6 May 2016
I thought birds were the successors to dinosaurs.
https://me.yahoo.com/a/JxVN0eQFqtmgoY7wC1cZM44ET_iAanxHQmLgYgX_Zhn8#57cad · 6 May 2016
Just Bob · 6 May 2016
John Harshman · 6 May 2016
https://me.yahoo.com/a/JxVN0eQFqtmgoY7wC1cZM44ET_iAanxHQmLgYgX_Zhn8#57cad · 6 May 2016
Pierce R. Butler · 6 May 2016
stevaroni · 6 May 2016
Robert Byers · 7 May 2016
Robert Byers · 7 May 2016
Rolf · 7 May 2016
Seems Robert is lost way out in the woods here. It doesn't matter which of the advantaged ones survive. It follows from the advantage of being advantaged that they will have a higher reproductive success rate. That means that percentwise they will producing more desendants that the not-so-advantaged ones.
A process like that means a percentwise higher number of advantaged ones survive and produce offspring in the population. In the long run the less advantaged varity faces the risk of disappearance, swamped by the advantaged ones.
It goes without saying that there always will be an element of "luck" and chance in reproduction within a population but that doesn't eliminate the effect of differential reproductive success due to genetic differences.
There is no picking going on, there is a process of survival and propagation going on.
harold · 7 May 2016
harold · 7 May 2016
Ron Okimoto · 7 May 2016
There is one simple observation that if any IDiot can read this should be understandable to any such IDiot.
Why didn't this guy ever publish and become as famous as Fisher and Wright?
He would really have something if he could demonstrate that he submitted such papers, but they were always rejected. It would be even better if he posted the reviewers comments and could demonstrate that they were incorrect or off topic.
It is really unbelievable to me that anyone could even have second thoughts on the situation. We have genetic theory put forward over 80 years ago, and this guy thinks that something is wrong with it, but never tried to demonstrate it.
Now, when his only audience are the cretins that are still IDiots when the bait and switch has been going down for over 14 years and not a single IDiot has ever gotten the promised ID science when they have needed it, he makes his claims. Who would believe the guy?
Really, the ISCID died 8 years ago, and they stopped publishing their "science" journal in 2005. That was supposed to be the IDiot science organization, and my bet is that this guy was a member, and he never presented this stupidity to his IDiot peers, or even they laughed him down.
Whenever the guys that sold the ID scam need to put forward the science of intelligent design all any IDiot ever gets is a switch scam that doesn't even mention that ID ever existed. Every single time with not a single exception for nearly a decade and a half. There never was any ID science worth discussing, so what is the draw, and why would anyone take anything like this seriously? What possible legitimate excuse could anyone have at this point in the history of the creationist ID scam?
harold · 7 May 2016
quentin-long · 7 May 2016
Pierce R. Butler · 7 May 2016
I activate the "widgets" (a term Apple stole from small developers who couldn't fight back) by moving the pointer to a (user-selectable) corner, so stevaroni's directions - though including extra steps unnecessary on a Mac - could apply in a broad sense.
As stevaroni points out, Apple's calculator is pretty light-duty - but then, so is the number-crunching required in the original post here.
harold · 7 May 2016
robert van bakel · 7 May 2016
I don't think I've been banned, although I haven't posted in a while. Please don't tell me we're taking a leaf out of UD's posting manifesto with a long moderation que.
robert van bakel · 7 May 2016
Apolgies for the unfounded accusation. I posted on the previous article and all is well; my mistake!
Robert Byers · 7 May 2016
Tom English · 7 May 2016
Mike Elzinga · 8 May 2016
Rolf · 8 May 2016
What the IDiots are saying and maybe even believe is that the universe is a barren, sterile "creation" that needs a deity to operate in the same way that a car needs someone with a foot on the accelerator.
They are a fifth column at the heels of science.
TomS · 8 May 2016
Ron Okimoto · 8 May 2016
harold · 8 May 2016
TomS · 8 May 2016
harold · 8 May 2016
harold · 8 May 2016
Scott F · 8 May 2016
TomS · 8 May 2016
harold · 8 May 2016
eric · 8 May 2016
Robert Byers · 8 May 2016
Dave Luckett · 8 May 2016
Uh... guys, language pedant hat on.
"Consistency" means the property of holding two or more propositions at the same time in the same way without logical contradiction. (It also means the physical graininess of a substance, smooth to rough, but that need not concern us here.)
A word that means "a group of people who provide political support" is "constituency".
Language pedant hat now removed. Carry on.
https://me.yahoo.com/a/TmT6tr96j8I7z.NSXVrs5i9QwNXEtw--#1813f · 8 May 2016
Hi: I differ in understanding the points at stake.
Natural Selection is a general functioning terminology, that generally applies.
But the particulars are quite the contrary to Natural Selection.
For example GENE POOL, interbreeding, does not rely on Natural Selection as such. Though a stronger male of degraded GENE Pool might win out in breeding, this male is already in a downward road, that NATURAL SELECTION is not going to improve...!!!
The HUMAN GENE POOL is also on a downward road...!
SO the QUESTION would be rather, how does NATURAL SELECTION "create" a improved GENE POOL...! And we are not talking about the BIBLE or any RELIGION...!
Cheers,
George.
purelogic.
Dave Luckett · 9 May 2016
Your difficulty, 813f, lies in your definition of "stronger", as in "stronger male". Larger, more physically powerful males only succeed differentially where the environment selects for strength and size. It often does not. Metabolic economy, dexterity, light weight (as in species that fly or climb or use extended tree limbs), efficiency in food gathering or feeding (large teeth might detract, however impressive they are), speed or endurance or hardiness (often opposed by size or mass), reproductive efficiency (how good is your sperm at defeating the sperm of other males), sexual selection by the female, the relative success of opposed mating strategies, and so on. All these, and many other characteristics, have nothing to do with "strength", but they may - and do - produce greater "fitness".
That is how natural selection creates an improved gene pool - by selecting the characteristics that actually succeed in the extant environment. That environment includes many other species, and other members of the same species. And no, we are not talking about the Bible. Or at least, I'm not.
I can only guess at what you mean by "Though a stronger male of degraded GENE Pool might win out in breeding, this male is already in a downward road, that NATURAL SELECTION is not going to improveâ¦!!!" Does "this male" mean you? If so, I can only infer that your complaint is that you're not getting any. My commiserations, if that is the case.
Dave Luckett · 9 May 2016
Oh, I see. Sorry. I misconstrued 813f. He means, I think, that a male of "degraded GENE pool" (sic) might have greater reproductive success, even though he is on a "downward road".
No. Wrong. Statistically, the male who enjoys greater reproductive success does so because he has better genes, not "degraded" ones. Better at thriving in the environment, that is. The whole point made by population genetics, as the head post demonstrates, is that despite the occasional reproductive success of individuals (not only males) who have less successful genetic traits, any genetic advantage in a given environment, however small, will create greater reproductive success and will rapidly diffuse through a population.
harold · 9 May 2016
eric · 9 May 2016
Dave Luckett · 9 May 2016
TomS · 9 May 2016
Joe Felsenstein · 9 May 2016
Michael Fugate · 9 May 2016
eric · 9 May 2016
Richard Bond · 9 May 2016
For all the loyal Robert Byers fans at this site, I have to concede that, for once, he has a point, in his first post in this thread. Mootoo Kimura showed in a classic paper (http://www.well.ox.ac.uk/~gerton/Gulbenkian/kimura-diffusion.pdf) that even advantageous genes are unlikely to become established. In an admittedly simplified model, he calculated the probability of fixation as 2s/(1-exp(-4Ns)), where s is the selective advantage. For a large population, and small s, that means that the probability of a gene reaching fixation is 2s. For s=0.01, quoted in Joe Felsenstein's post, the probability is only 0.02. The calculations that demonstrate the inexorable progress of even a quite small advantage only come into play once the gene has spread through quite a large proportion of the population.
Michael Fugate · 9 May 2016
0.02 is 1 in 50 - not bad odds. Better than Leicester City winning the EPL!
Joe Felsenstein · 9 May 2016
I don't think Robert Byers made a valid point here. (It was, however on topic so I did not exile it to the Wall).
Yes, the fixation probability Kimura calculated for a single copy was (1-exp(-2s))/(1-exp(-4Ns)) which for N = 10,000 and s = 0.01 works out to 0.0198013 (2s, which is Haldans's 1927 approximation, is near that value).
But if the allele is instead neutral, the fixation probability is 1/20,000 which is 0.00005. That is more than 396 times smaller. So natural selection has a big effect, in spite of what Byers says.
We have been this way before, with the role of Byers played by Salvador Cordova. See my post on this at PT: here.
TomS · 9 May 2016
harold · 9 May 2016
Joe Felsenstein · 9 May 2016
Robert Byers · 9 May 2016
Robert Byers · 9 May 2016
Robert Byers · 9 May 2016
phhht · 9 May 2016
Daniel · 9 May 2016
Malcolm · 10 May 2016
DanHolme · 10 May 2016
Joe Felsenstein · 10 May 2016
Byers is raising the issue of species that are divided into multiple populations, with fitnesses that differ in different populations. He imagines that this prevents advantageous mutations from being incorporated.
Back in the 5th comment on this thread, in response to eric, I mentioned that there is a substantial literature in theoretical population genetics on natural selection that varies in time and space. In fact, in 1976, a mere 40 years ago, I published a review article on this literature, and I managed to cover 152 theoretical papers. Since then four decades have elapsed -- the literature has probably at least tripled.
I think that I will reserve comment on the general issue until Robert has read more of this literature. Right now I think he has read approximately zero of these papers.
Robert Byers · 10 May 2016
Robert Byers · 10 May 2016
eric · 11 May 2016
DS · 11 May 2016
Well my gut feeling is that booby will never be right about anything. Therefore, if I follow my gut feeling, all of his incoherent blubbering can be dismissed as ignorant nonsense. So, according to the logic being used by booby himself, he should be ridiculed or ignored. My intuition tells me that someone who refuses to learn proper grammar and spelling will never learn math either. My intuition tells me that, if the house edge in blackjack is 0.28% and the house almost always wins, that the odds of booby ever understanding the argument are much less than 0.28%. That and he should stay out of casinos, if he knows what is good for him.
Like Mikey, booby should come back when he has read the relevant literature. Maybe then someone will care what he thinks.
Just Bob · 11 May 2016
Robert, the whole human race's instinct told us for almost all of history that the Earth does not move, and that the sun, moon, and stars travel around our unmoving Earth, which is at the center of the solar system and the entire universe. That's when all of humanity was on "your side," including Bible writers.
That instinct was WRONG. Some people still go with that instinct, and some of them do it because that's what the Bible tells them. But they're still WRONG.
So what do you think? Did our instinct that the Earth doesn't move mislead us for all that time? Or do you go with your instinct (and the Bible) on that, too? Is astronomy also "atomic and unproven"?
Rolf · 11 May 2016
eric · 11 May 2016
Malcolm · 11 May 2016
Robert Byers · 11 May 2016
Scott F · 11 May 2016
Robert Byers · 11 May 2016
Joe Felsenstein · 11 May 2016
Rolf · 12 May 2016
From now on, I'll pretend Robert Byers doesn't exsist. I think all his posts should default to the Wall, except maybe his first in a thread to forestall censorship claims.
He's only a Drosophila in the ointment.
eric · 12 May 2016
TomS · 12 May 2016
Rolf · 12 May 2016
I concur. I found an escape hatch with My Dear Aunt Sally, and parantheses. I assume that would be too much for Robert.
Bobsie · 12 May 2016
As a layman, this is my understanding.
Yes, of course in a single generation a beneficial mutation is chump change compared to the randomness throughout the gene pool. However, the power of natural selection becomes apparent as it gets magnified through successive generations.
Here's how: The randomness is always still there but itâs always different and never the same in subsequent generations, thatâs the essence of ârandomnessâ. On the other hand, the beneficial mutation plays the same advantage again and again in each subsequent generation bestowing the same advantage to an ever increasing proportion of the gene pool and eventually overwhelming the effect of any random noise variation.
Bobsie · 12 May 2016
Layman addendum: If the insect with the beneficial mutation that smashed against your windshield had not yet reproduced, then there is no beneficial mutation in the gene pool.
However, if that insect already had offspring, and that offspring had a minuscule reproductive advantage due to one or more of any circumstances; environment, biology, behavior, prey or predator to name a few; unless circumstances change again, the benefit derived from the mutation cannot be denied the gene pool. Itâs inevitable. Success breeds success.
Malcolm · 12 May 2016
Malcolm · 12 May 2016
Eric Finn · 12 May 2016
Robert Byers · 12 May 2016
Robert Byers · 12 May 2016
Robert Byers · 12 May 2016
malcome.
You say its not the first generation that must die but IT must be eventually the whole population dies otherwise the few advantaged are always a trivial minority.
Evolutionary genetics is not based on casino math.
its based on a necessary oTHER event going on. the demise of the population without the advantage. It doesn't matter how many generations and indeed your predicting generations can keep going but are in decline for some reason. Why/ Until the decline there is no impact on that population and no evolution.
I think everyone here missed something.
Mutations will not change a population/evole it based on minor breeding events.Its essential the oTHER SIDE does not breed.
if it does breed there is no evolution of that type of creature.
So instinct rightly kicks in that too few won't change things.
Thats the point from the DI scientist.
Your side dOES need extinction and right quick.
Robert Byers · 12 May 2016
https://me.yahoo.com/a/TmT6tr96j8I7z.NSXVrs5i9QwNXEtw--#1813f · 12 May 2016
https://me.yahoo.com/a/TmT6tr96j8I7z.NSXVrs5i9QwNXEtw--#1813f · 12 May 2016
813f
Hi. No doubt that wishful thinking and babbling big words and no reality is as easy as your ill fetched Evolution!
I do not believe in Evolution as such nor in JESUS! As I do not believe in âmagicâ from a dead MOTHER NATUREâ¦!!! In logic and purelogic all physical or matter should have a known logic explanation or a not known one beyond our minds!
But to infer Evolution directly because your small brain cannot explain a God or gods or entities, is proof or your small Intelligence sourceâ¦!
http://anagrammatt3.blogspot.ca/
But believe Science has been assaulted by MAGICIANS and ALCHEMISTS, and you name whatâ¦!
INTERBREEDING, implies depleted numbers, etcâ¦! What does it cause? IMPROVEMENT in GENETICSâ¦???
Ohâ¦! You guys talk bigger than your head or shoes!
Cheers. Ask Mother Nature to better your minds intelligence!
George.
Dave Luckett · 12 May 2016
Here's a big word, George: intelligibility. Look it up.
Eric Finn · 12 May 2016
Malcolm · 13 May 2016
TomS · 13 May 2016
eric · 13 May 2016
eric · 13 May 2016
Bobsie · 13 May 2016
gnome de net · 13 May 2016
Joe Felsenstein · 13 May 2016
Many others have done a good job of answering Robert Byers when he says that, unless there is a great die-off of others, a beneficial mutant will not spread.
I do just want to mention that Byers's mistake is also committed by many other creationists and ID supporters. They often say that natural selection cannot create, it can only destroy, It eliminates the less fit but does not increase the more fit. There are two problems with this:
1. It ignores the role of differential fertility, as it assumes that all fitness differences are differential viability, and
2. It commits Byers's fallacy. It ignores the change of the frequencies of genotypes as the less fit are eliminated.
When the genotype frequencies change, and with them the gene frequencies, the composition of the population changes. Even if there were only one generation of this change, future gene frequencies would reflect the change.
These folks do not understand that populations generally have a excess reproduction. They seem never to have heard of density-dependent population size regulation. Eliminating the less viable does mean that, in the presence of density regulation, the more viable will increase. Not only in frequency, but also in actual numbers.
And of course this has been discussed, all the way back to Darwin, who made it an essential part of his arguments.
TomS · 13 May 2016
Just Bob · 13 May 2016
Robert, do you know why we had to invent science? It was because our "instincts" were so often WRONG. Instinct couldn't tell us that the Earth rotates, or that there are micro-organisms that make you sick, or that smaller objects fall as fast as large ones, or that matter is made of atoms, or that that little static electric spark was something that could power a whole civilization -- and allow you to complain on the internet about how your instinct says science is wrong.
Flint · 13 May 2016
I keep getting the impression that Byers is visualizing competition among seeds, or the seed-equivalent of any species whose reproductive strategy is to crank out enormous numbers of potential reproducers, almost none of which will ever reach the breeding stage. He doesn't seem to grasp that the selection isn't between seeds, but between breeding adults. And that the adults that tend to survive to breed (if this occurred because they are of a variety that gives them a very slight edge) will then generate enormous numbers of seeds ALL of which possess this favorable variant. And if they all have it, it really doesn't matter which of them randomly reach the breeding stage.
Scott F · 13 May 2016
Robert Byers · 13 May 2016
Scott F · 13 May 2016
Robert Byers · 13 May 2016
Robert Byers · 13 May 2016
PA Poland · 13 May 2016
Daniel · 14 May 2016
TomS · 14 May 2016
One thing which I disagree with you on.
Any mathematics at all is off-putting for very many people. Even middle-school level math.
That is why people patronize casinos, why they accept huge interest rates, etc. etc.
Bobsie · 14 May 2016
Bobsie · 14 May 2016
Mr. Byers, your "original" group doesn't ever die off; they all eventually "marry" off and join the successful "new" clan.
https://me.yahoo.com/a/TmT6tr96j8I7z.NSXVrs5i9QwNXEtw--#1813f · 14 May 2016
https://me.yahoo.com/a/TmT6tr96j8I7z.NSXVrs5i9QwNXEtw--#1813f · 14 May 2016
Author Profile Page A Masked Panda (813f) replied to a comment from Dave Luckett | May 14, 2016 11:21 AM | Reply
http://pandasthumb.org/archives/2016/05/a-devastating-c.html#comment-353005
Dave Luckett said:
Hereâs a big word, George: intelligibility. Look it up.
Oh!
Yes, I looked up intelligibility, and it says something about SIGN LANGUAGE and ANALPHABETISM!
Can somebody explain, after SELF-developing REPRODUCTION methods, then the CELL increases in complexity, to the biggest GENOMICS, accepted of THE SALAMANDER?
ALSO rubbish DNA, is not a ânegative loadâ, but useful?
So there were not enough numbers to better genetics, if that is a statement.
So how do you make billions of numbers to occur? That is what must have happened is what you guys are presumptively saying!
FIRST LETS MAKE LIFE IN A LABORATORY, THEN TELL YOUR SCIENCE EVOLUTION AND RELIGION âSTORIESââ¦! MAGICIANS!
George. Pure Logic and Logic. http://www.dictionary.com/browse/presumptively
Just Bob · 14 May 2016
Robert, you have a competitor. Actually I can make more sense out of your writing than George's.
And you don't yell in our faces with CAPS LOCK in random places.
Dave Luckett · 14 May 2016
George is either a raging loony, or he's imitating one. It's impossible to say which, but my instinct is that he might be a bit too good to be true.
gnome de net · 14 May 2016
@ Robert Byers
You continue to insist that the math doesnât work for evolution's influence on population genetics; that the math must add up.
We're still waiting for you to show us that failed math.
Robert Byers · 14 May 2016
Robert Byers · 14 May 2016
D Stowens · 14 May 2016
DaveH · 15 May 2016
Rolf · 15 May 2016
With or without evolution, the genomics within a population are in a constant flux.
A population is not a fixed thing, each day sees a new population replacing the old one. Births and deaths at work 24/7. They are what does the work, genomics have a free ride.
DS · 15 May 2016
Robert,
If a beneficial dominant allele arises by mutation and the selection coefficient for homozygous recessives is 0.01, how many generations will be required for fixation of the new allele?
Look up the proper equation, do the math and tell us the answer. When you do, you will see how evolution works. Until you do, your intuition is worthless. You can go on ignoring the experts, that isn't gong to do you any good. You can go on in ignorance, ranting and raving that something is wrong somewhere, but that is not going to do you any good. You can go on thrashing up this thread for the next twenty pages, but that isn't going to do you any good either. Put up or shut up.
harold · 15 May 2016
TomS · 15 May 2016
TomS · 15 May 2016
harold · 15 May 2016
harold · 15 May 2016
To imply that emergence of a new adaptation forces the extinction of ancestors would not only be to commit the "why are there still monkeys" fallacy, but also, would raise the question "why is there more than one species on Earth", as, if every time a beneficial allele arose, it rapidly became fixed and the ancestral phenotype rapidly became extinct, there would be one or two species at any given time, although never the same species for long.
harold · 15 May 2016
Nothing I am saying is in dispute with the basic points made by Joe Felsenstein or others, including PA Poland, nor should any agreement with the many very, very wrong statements made by Byers in this thread be imputed to me. Some of Byers' early points amounted to denial of math that has been proven since the 18th century, although he seems to have moved away from that as the thread progressed.
A lot of it comes down to what we define as the population.
If all individuals are still considered members of the population, then of course the frequency of all other alleles at that locus must decrease for the new beneficial allele at that locus to increase in frequency. (This is true regardless of changes in absolute population size.)
If we allow for new populations branching off of the old population, the new allele may come to define a new population.
Ravi · 15 May 2016
DS · 15 May 2016
Just Bob · 15 May 2016
Joe Felsenstein · 15 May 2016
Robert Byers · 15 May 2016
Robert Byers · 15 May 2016
Robert Byers · 15 May 2016
Robert Byers · 15 May 2016
Robert Byers · 16 May 2016
Ravi · 16 May 2016
DS · 16 May 2016
Ravi · 16 May 2016
DS · 16 May 2016
DS · 16 May 2016
Joe Felsenstein · 16 May 2016
Ravi · 16 May 2016
DS · 16 May 2016
Sorry if that link does not work for you. Here is the reference:
Grant and Flake (1974) Solutions to the cost of selection dilemma. PNAS 71(10):3863-5.
And I only hit submit once.
Joe Felsenstein · 16 May 2016
eric · 16 May 2016
Joe Felsenstein · 16 May 2016
In effect, what Ravi is saying is that if you and the other runner are successfully outrunning the bear, then neither runner will get ahead of the other.
Ravi · 16 May 2016
Joe Felsenstein · 16 May 2016
Wrong. Population geneticists model all kinds of cases. I know. I compiled the only bibliography ever compiled for the field.
See above comment by me about the two runners who are outrunning the bear.
eric · 16 May 2016
eric · 16 May 2016
Just Bob · 16 May 2016
Ravi, before you try to teach population genetics to scientists, see if you can teach Robert Byers to spell huge.
Michael Fugate · 16 May 2016
Why let facts get in the way of belief?
Michael Fugate · 16 May 2016
Ravi's and Robert's motto: "Why let facts get in the way of belief?"
prongs · 16 May 2016
eric · 16 May 2016
Just Bob · 16 May 2016
https://me.yahoo.com/a/TmT6tr96j8I7z.NSXVrs5i9QwNXEtw--#1813f · 16 May 2016
https://me.yahoo.com/a/TmT6tr96j8I7z.NSXVrs5i9QwNXEtw--#1813f · 16 May 2016
Particularly I still do not understand MEOSIS and MITOSOS! Nor chromosomes either! So ALELES is like pushing it!
No doubt good traits in a population, are expected to take first stage in the process.
Though in my case, my offspring are of lower genetic degree!
Can you explain this for me please?
George - Pure Logic and Logic.
https://me.yahoo.com/a/TmT6tr96j8I7z.NSXVrs5i9QwNXEtw--#1813f · 16 May 2016
sorry. MITOSIS!
Just Bob · 16 May 2016
Just Bob · 16 May 2016
George, do you acknowledge that professional scientists probably know what they're talking about in their own fields of study? And are they more likely or less likely to be correct about facts in their fields than a non-scientist?
If you don't believe what biologists tell us about biology, why not?
Michael Fugate · 16 May 2016
Ravi · 16 May 2016
Ravi · 16 May 2016
eric · 16 May 2016
Joe Felsenstein · 16 May 2016
Michael Fugate · 16 May 2016
To reiterate, if one looks at population genetics models - one will find that mutation, migration, population size, heritability, selection, etc. can be included. Even if selection is weaker than any or all of the other factors, selection is still acting and the result is different than if selection coefficient were = 0.
Robert Byers · 16 May 2016
prongs · 16 May 2016
Just Bob · 16 May 2016
Robert, did you just retire, so now you can turn your full attention to explaining to all the scientists why they're wrong about all the science they've studied and researched over long careers?
Do you have any other hobbies?
Or is this maybe a Mission from God?
https://me.yahoo.com/a/TmT6tr96j8I7z.NSXVrs5i9QwNXEtw--#1813f · 16 May 2016
https://me.yahoo.com/a/TmT6tr96j8I7z.NSXVrs5i9QwNXEtw--#1813f · 16 May 2016
Just Bob · 16 May 2016
Yep, sorry I asked, and no wiser.
Malcolm · 16 May 2016
Joe Felsenstein · 17 May 2016
George Purelogic (Masked Panda 813f) : If you doubt the conclusions of science with regard to meiosis and mitosis, a matter which was settled 100 years ago to the satisfaction of almost all scientists, and hasn't gotten any more uncertain since, please take the matter to some other forum. It is off-topic to this thread, and even off-topic to this whole forum.
I am moderating this thread, and will send further discussion of this issue to PT's Bathroom Wall. You don't want to go there, really.
And that holds for any replies to Purelogic on that issue -- they go to the BW too, to help discourage troll-chasing as well as trolling.
Ravi · 17 May 2016
Ravi · 17 May 2016
Michael Fugate · 17 May 2016
Just Bob · 17 May 2016
Ravi, perhaps you could explain how or why there could be a "population where there is little or no reproductive excess."
In times of opportunity, such a species could not expand to take advantage of new territory or resources. In times of stress, when some potential breeders die before breeding, then the population would rapidly diminish and head for extinction. Excess breeders and offspring are a species' insurance against hard times.
And finally, how could a "population where there is little or no reproductive excess", say one with millions of individuals, ever reach a population of millions, if "there is little or no reproductive excess"?
Joe Felsenstein · 17 May 2016
Joe Felsenstein · 17 May 2016
Joe Felsenstein · 17 May 2016
Actually you can just computer-simulate that using the teaching program PopG (available here) which I distribute for use in courses teaching population genetics.
Ravi can try that and report the results here.
eric · 17 May 2016
Robert Byers · 17 May 2016
My final post on this is my last hunch.
It occurs to me that the whole concept of there being a beneficial gene and so a advantageous individuals is another rub.
Just having or saying there is this advantage and so reproductive advantage is a interference in the original population.
I mean WHY is there a advantage? why at all? If the original pop is thriving and hugh wHY should there EVER be this benificial group interfering.
I see in this the flaw of the evolutionists on this issue/thread.
This , I think, is the complain from the DI.
Its not a math race but why is there a race.
In reality there never would be a beneficial gene/reproducing individuals. This wouldn't happen in these high pops of creatures.
Its an abstract concept that a real scientist dealing with mutations rejected.
A better way he could put it IS that a mutation would never bring a benefit for reproduction in a gugh/thriving group.
Thats the rub.
Malcolm · 17 May 2016
Just Bob · 17 May 2016
Sorry, Robert, but it's really hard to take you seriously when you go "gugh".
I mean, the stuff you say is ignorantly wrong, but "hugh" and "gugh" make it hard not to burst out laughing.
Dave Luckett · 18 May 2016
Malcolm · 18 May 2016
Michael Fugate · 18 May 2016
"Saying that it isnât possible to have a reproductive advantage in a large population is pretty moronic, even for Byers."
Kind of like saying it isn't possible to have a sales advantage in the fizzy drinks market...
eric · 18 May 2016
Gthe tooth fairy.Robert Byers · 18 May 2016
gnome de net · 18 May 2016
Joe Felsenstein · 18 May 2016
He's in the great tradition of opera singers who give annual "farewell tours".
Michael Fugate · 19 May 2016
Scott F · 19 May 2016
eric · 20 May 2016
Scott F · 21 May 2016