Most of you don't understand evolution. I mean this in the most charitable way; there's a common conceptual model of how evolution occurs that I find everywhere, and that I particularly find common among bright young students who are just getting enthusiastic about biology. Let me give you the Standard Story, the one that I get all the time from supporters of biology.
Evolution proceeds by mutation and selection. A novel mutation occurs in a gene that gives the individual inheriting it an advantage, and that person passes it on to their children who also gets the advantage and do better than their peers, and leave more offspring. Given time, the advantageous mutation spreads through the population so the entire species has it.
One example is the human brain. An ape man millions of years ago acquired a mutation that made his or her brain slightly larger, and since those individuals were slightly smarter than other ape men, it spread through the population. Then later, other mutations occured and were selected for and so human brains gradually got larger and larger.
You either know what's wrong here or you're feeling a little uneasy—I gave you enough hints that you know I'm going to complain about that story, but if your knowledge is at the Evolutionary Biology 101 level, you may not be sure what it is.
Just to make you even more queasy, the misunderstanding here is one that creationists have, too. If you've ever encountered the cryptic phrase "RM+NS" ("random mutation + natural selection") used as a pejorative on a creationist site, you've found someone with this affliction. They've got it completely wrong.
Here's the problem, and also a brief introduction to Evolutionary Biology 201.
First, it's not exactly wrong — it's more like taking one good explanation of certain kinds of evolution and making it a sweeping claim that that is how all evolution works. By reducing it to this one scheme, though, it makes evolution far too plodding and linear, and reduces it all to a sort of personal narrative. It isn't any of those things. What's left out in the 101 story, and in creationist tales, is that: evolution is about populations, so many changes go on in parallel; selectable traits are usually the product of networks of genes, so there are rarely single alleles that can be categorized as the effector of change; and genes and gene networks are plastic or responsive to the environment. All of these complications make the actual story more complicated and interesting, and also, perhaps to your surprise, make evolutionary change faster and more powerful.
Think populations
Mutations are the root of biological variation, of course, but we often have a naive view of their consequences. Most mutations are neutral. Even advantageous mutations are subject to laws of chance in their propagation, and a positive selection coefficient does not mean there will be an inexorable march to fixation, where every individual has the allele. This is also true of deleterious mutations: chance often dominates, and unless it is a strongly negative allele, like an embryonic lethal mutation, there's also a chance it can spread through the population.
Stop thinking of mutations as unitary events that either get swiftly culled, because they're deleterious, or get swiftly hauled into prominence by the uplifting crane of natural selection. Mutations are usually negligible changes that get tossed into the stewpot of the gene pool, where they simmer mostly unnoticed and invisible to selection. Look at human faces, for instance: they're all different, and unless you're looking at the extremes of beauty or ugliness, the variations simply don't make much difference. Yet all those different faces really are the result of subtly different combinations of mutant forms of genes.
"Combinations" is the magic word. A single mutation rarely has a significant effect on a feature, but the combination of multiple mutations may have a detectable or even novel effect that can be seen by natural selection. And that's what's going on all the time: the population is a huge reservoir of genetic variation, and what we do when we reproduce is sort and mix and generate new combinations that are then tested in the environment.
Compare it to a game of poker. A two of hearts in itself seems to be a pathetic little card, but if it's part of a flush or a straight or three of a kind, it can produce a winning hand. In the game, it's not the card itself that has power, it's its utility in a pattern or combination of other cards. A large population like ours is a great shuffler that is producing millions of new hands every day.
We know that this recombination is essential to the rapid acquisition of new phenotypes. Here are some results from a classic experiment by Waddington. Waddington noted that fruit flies expressed the odd trait of developing four wings (the bithorax phenotype) instead of two if they were exposed to ether early in development. This is not a mutation! This is called a phenocopy, where an environmental factor induces an effect similar to a genetic mutation.
What Waddington did next was to select for individuals that expressed the bithorax phenotype most robustly, or that were better at resisting the ether, and found that he could get a progressive strengthening of the response.

The progress of selection for or against a bithorax-like response to ether treatment in two wild-type populations. Experiments 1 and 2 initially showed about 25 and 48% of the bithorax (He) phenotype.
This occurred over 10s of generations — far, far too fast for this to be a consequence of the generation of new mutations. What Waddington was doing was selecting for more potent combinations of alleles already extant in the gene pool.
This was confirmed in a cool way with a simple experiment: the results in the graph above were obtained from wild-caught populations. Using highly inbred laboratory strains that have greatly reduced genetic variation abolishes the outcome.
Jonathan Bard sees this as a powerful potential factor in evolution.
Waddington's results have excited considerable controversy over the years, for example as to whether they reflect threshold effects or hidden variation. In my view, these arguments are irrelevant to the key point: within a population of organisms, there is enough intrinsic variability that, given strong selection pressures, minor but existing variants in a trait that are not normally noticeable can rapidly become the majority phenotype without new mutations. The implications for evolution are obvious: normally silent mutations in a population can lead to adaptation if selection pressures are high enough. This view provides a sensible explanation of the relatively rapid origins of the different beak morphologies of Darwin's various finches and of species flocks.
Think networks
One question you might have at this point is that the model above suggests that mutations are constantly being thrown into the population's gene pool and are steadily accumulating — it means that there must be a remarkable amount of genetic variation between individuals (and there is! It's been measured), yet we generally don't see most people as weird and obvious mutants. That variation is largely invisible, or represents mere minor variations that we don't regard as at all remarkable. How can that be?
One important reason is that most traits are not the product of single genes, but of combinations of genes working together in complex ways. The unit producing the phenotype is most often a network of genes and gene products, such at this lovely example of the network supporting expression and regulation of the epidermal growth factor (EGF) pathway.
That is awesomely complex, and yes, if you're a creationist you're probably wrongly thinking there is no way that can evolve. The curious thing is, though, that the more elaborate the network, the more pieces tangled into the pathway, the smaller the effect of any individual component (in general, of course). What we find over and over again is that many mutations to any one component may have a completely indetectable effect on the output. The system is buffered to produce a reliable yield.
This is the way networks often work. Consider the internet, for example: a complex network with many components and many different routes to get a single from Point A to Point B. What happens if you take out a single node, or even a set of nodes? The system routes automatically around any damage, without any intelligent agency required to consciously reroute messages.
But further, consider the nature of most mutations in a biological network. Simple knockouts of a whole component are possible, but often what will happen are smaller effects. These gene products are typically enzymes; what happens is a shift in kinetics that will more subtly modify expression. The challenge is to measure and compute these effects.
Graph analysis is showing how networks can be partitioned and analysed, while work on the kinetics of networks has shown first that it is possible to simplify the mathematics of the differential equation models and, second, that the detailed output of a network is relatively insensitive to changes in most of the reaction parameters. What this latter work means is that most gene mutations will have relatively minor effects on the networks in which their proteins are involved, and some will have none, perhaps because they are part of secondary pathways and so redundant under normal circumstances. Indirect evidence for this comes from the surprising observation that many gene knockouts in mice result in an apparently normal phenotype. Within an evolutionary context, it would thus be expected that, across a population of organisms, most mutations in a network would effectively be silent, in that they would give no selective advantage under normal conditions. It is one of the tasks of systems biologists to understand how and where mutations can lead to sufficient variation in networks properties for selection to have something on which to act.
Combine this with population effects. The population can accumulate many of these sneaky variants that have no significant effect on most individuals, but under conditions of strong selection, combinations of these variants, that together can have detectable effects, can be exposed to selection.
Think flexible genes
Another factor in this process (one that Bard does not touch on) is that the individual genes themselves are not invariant units. Mutations can affect how genes contribute to the network, but in addition, the same allele can have different consequences in different genetic backgrounds — it is affected by the other genes in the network — and also has different consquences in different external environments.
Everything is fluid. Biology isn't about fixed and rigidly invariant processes — it's about squishy, dynamic, and interactive stuff making do.
Now do you see what's wrong with the simplistic caricature of evolution at the top of this article? It's superficial; it ignores the richness of real biology; it limits and constrains the potential of evolution unrealistically. The concept of evolution as a change in allele frequencies over time is one small part of the whole of evolutionary processes. You've got to include network theory and gene and environmental interactions to really understand the phenomena. And the cool thing is that all of these perspectives make evolution an even more powerful force.
Bard J (2010) A systems biology view of evolutionary genetics. Bioessays 32: 559-563.


106 Comments
MrG · 24 July 2010
Good stuff.
GuessWho · 24 July 2010
Stephen · 24 July 2010
Thanks for clarifying it, I'll be sure to send my partially retarded Xians here the next time I have to deal with someone who doesn't understand evolution. I know they won't actually read it, but they also won't listen to me, so why bother wasting my time trying to explain it?
MrG · 24 July 2010
DS · 24 July 2010
This is exactly why the argument from ignorance and incredulity fails so miserably. An uninformed person can easily dismiss a simplified and fundamentally flawed notion. It takes an expert to understand all of the subtleties that go into modern evolutionary theory. FIrst, you must understand basic genetics. Then you must understand mutations. Then you must understand population genetics. Then you must understand developmental genetics. And when you have mastered all of that you can finally begin to grasp some of the complexities involved. That's why uninformed people and people who are not equipped to look at evidence, or who simply refuse to look at evidence, are not entitled to an opinion. That is why we have experts in all of these fields. That is why we should listen to the experts. Blindly dismissing the experts, because you in your own ignorance cannot imagine how something could work, is simply arrogance.
Mike Elzinga · 24 July 2010
I really like PZ’s explanations. The more we see the underlying details, the more they tie the processes of evolution to the physics and chemistry.
In the physics of complex systems subjected to their surrounding environments, it is often the emergent properties that guide subsequent stages of development within those environments.
Those emergent properties are usually unpredictable (before the fact) characteristics that are manifestations of myriads of underlying physical and chemical processes.
A nice example of selection of emergent features would be the stockiness of the limbs of ambulatory creatures in a gravitational field. As structures get bigger, weight becomes important and gravity becomes an environmental “pressure.” The strength of the emergent limb is proportional to the square of its dimension, i.e., its cross-sectional area. But weight is proportional to the cube of the dimensions of the creature, i.e., its volume.
Those morphological features are both emergent properties that are the result of many underlying processes, no particular one of which is the sole determiner of those gross features. But once those gross features become significant in a gravitational field, they become features which can be modified by selection in subsequent populations.
I like to think of a chain of populations as analogous to a single system that makes its adjustments to surrounding “pressures” by using time-sequenced surrogates of itself. In other words, if one were to take snapshots of successive adult representatives of a species throughout its evolutionary history and play them back in succession, it would look like a single individual changing through time.
A single system “relaxes into” its current potential wells by minimizing its potential energy in the most general sense. Reproducing systems do this through slightly morphed surrogates in successive generations.
Dale Husband · 24 July 2010
Mike Elzinga · 24 July 2010
robert van bakel · 25 July 2010
Actually, this PZ blog was oddly reassuring to me. I always knew that RM-NS worked, and I always knew it worked while I was asleep, awake, or not paying attention. This is not 'rocket science', it is infinately more intricate, and strangely, comprehensible.
Changes occur in populations, advantageous changes could lead to new phenotypes, this COULD give reproductive advantage, the environment could alter, the new advantage could become a disadvantage, the environment could change, new mutations (occuring as I sleep) COULD.... anyway you and I, and nature get the idea.
RBH · 25 July 2010
RBH · 25 July 2010
Oops. Read the OP before commenting, RBH.
Rusty Catheter · 25 July 2010
So, looking at all these neutral mutations of cistrons,the ones that only change the nucletide but not the amino acid, and the further ones that make conservative amino acid changes, we can summarise that many improved mutations are just waiting for the correct complements to come along.
Point mutations of introns and intercistronic sequences have more immediate effects. If we propose that these regulate cistronic activity by their binding of proteins *like* transcription factors and cis/trans factors, *and* their binding of RNA sequences (let us regard these too as transcription factors) potentially with their own point mutations.
.....then there is considerable scope to modulate the expression of the cistronic genome without any significant alteration of coding sequences.
Yet I think it is not fair to disparage the simplistic model. It is fine for single-gene traits, particularly in bacterial genetics.
Rusty
MrG · 25 July 2010
MartinC · 25 July 2010
"You've got to include network theory and gene and environmental interactions to really understand the phenomena."
You can include whatever you want - it will still remain the old darwinism in new clothes. The only difference is that this "network" enable darwinists to explain what is really observed in nature - sudden changes in morphology or saltationism. It was Richard Goldschmidt who observed and studied phenocopies on butterflies wings btw. As you may know this prominent geneticist dismissed darwinian model and coined the term "systemic mutation" to explain sudden changes observed in nature.
PZ Myers'"network" is now a new helper in this pseudo-scientifical narrative called neodarwinism. "Network" can somehow mysteriously accumulate enough "silent changes" only to expose them at the right moment as phenocopies or phenotypes - and doctor Myers obviously confound these two terms to support his "network" version of darwinism. Actually such "network" approach can inhibit evolution and not to promote it. See "Frozen evolution" or "Elastic not plastic evolution" by professor Flegr here:
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2823622/
Professor Flegr has mentioned also pleiotropy and epistatic interactions mentioned in PZMyers article using layman words. And so we can see - for one neodarwinist "network" can promote evolution and for another neodarwinist the same network inhibits evolution.
"It is one of the tasks of systems biologists to understand how and where mutations can lead to sufficient variation in networks properties for selection to have something on which to act."
Maybe better solution would be to issue a decree for "system biologists" how to interpret this mysterious "networks" in order to save neodarwinian narrative in the coming years.
Frank J · 25 July 2010
MartinC · 25 July 2010
"You've got to include network theory and gene and environmental interactions to really understand the phenomena."
You can include whatever you want - it will still remain the old darwinism in new clothes. The only difference is that this "network" enable darwinists to explain what is really observed in nature - sudden changes in morphology or saltationism. It was Richard Goldschmidt who observed and studied phenocopies on butterflies wings btw. As you may know this prominent geneticist dismissed darwinian model and coined the term "systemic mutation" to explain sudden changes observed in nature.
PZ Myers'"network" is now a new helper in this pseudo-scientifical narrative called neodarwinism. "Network" can somehow mysteriously accumulate enough "silent changes" only to expose them at the right moment as phenocopies or phenotypes - and doctor Myers obviously confound these two terms to support his "network" version of darwinism. Actually such "network" approach can inhibit evolution and not to promote it. See "Frozen evolution" or "Elastic not plastic evolution" by professor Flegr here:
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2823622/
Professor Flegr has mentioned also pleiotropy and epistatic interactions mentioned in PZMyers article using layman words. And so we can see - for one neodarwinist "network" can promote evolution and for another neodarwinist the same network inhibits evolution.
"It is one of the tasks of systems biologists to understand how and where mutations can lead to sufficient variation in networks properties for selection to have something on which to act."
Maybe better solution would be to issue a decree for "system biologists" how to interpret this mysterious "networks" in order to save neodarwinian narrative in the coming decades.
Pete Dunkelberg · 25 July 2010
MrG · 25 July 2010
DS · 25 July 2010
MartinC wrote:
"You can include whatever you want - it will still remain the old darwinism in new clothes. The only difference is that this “network” enable darwinists to explain what is really observed in nature - sudden changes in morphology or saltationism. It was Richard Goldschmidt who observed and studied phenocopies on butterflies wings btw. As you may know this prominent geneticist dismissed darwinian model and coined the term “systemic mutation” to explain sudden changes observed in nature.:
So evolution can sometimes proceed quickly and can sometimes proceed slowly. What exactly is your point? How is this a problem for evolutionary theory? Do you think that no real scientist has ever considered the effects of pleiotropy and epistasis? Do you honestly believe that these concepts are somehow a problem for modern evolutionary theory?
Here is a news flash for you, no one cares about "neodarwinism". Darwin was right about many things. Darwin was wrong about many things. So what? All that we have learned about modern genetics shows that evolution is a complicated process that must be examined on many different levels. Or do you think that "poof" is a better explanation?
Alan Fox · 25 July 2010
MrG · 25 July 2010
DS · 25 July 2010
Well, Martin still has not informed us of exactly what point he was trying to make. However, I did find this quote from the Flegr paper:
"It was argued above that adaptive evolution by means of Darwinian selection of individuals in a population, as well as by means of Dawkinsian competition between alleles in individual loci, is very difficult, if not impossible, in populations of sexual organisms. It is evident, however, that adaptive evolution operates on Earth and that this evolution is especially and extremely efficient in sexual organisms. It follows that a situation must occur, in which all three barriers to adaptive evolution are lifted, or at least temporarily operate with decreased efficiency."
So you can quibble about "neodarwinism" or "Dawkinsian competition" all you want, but evolution is still real and still works in all organisms, whether sexual or asexual.
Dale Husband · 25 July 2010
Mike Elzinga · 25 July 2010
Frank J · 25 July 2010
Elisheva Levin · 25 July 2010
Two comments:
1)I think the knowlege we have gained in the past 10 years about what actually happens inside cells with regard to gene expression (Immediate Early Genes, etc) have made the fact of evolution even clearer. The Biology 101 "explanation" is an example of an overgeneralization that leaves out important details. When dealing with enthusiastic beginning students, I always thought it important to bring in Hardy-Weinberg even at the beginning levels because it is so powerful to develop population genetics generalizations earl--even if the students are not ready for all the genetics details. In the '90's we waited until genetics (usually an upper 200 level course) to teach it. I have seen demo lessons to teach Hardy-Weinberg at the high school level conceptually, adding a short calculation for students who have the math skills.
2) I was a geologist before becoming a biologist, and I have some real problems with the current "climate science" that does not seen to take earth history into account. That global climate change has occurred in earth history is beyond dispute, but to claim that the earth is on a linear global warming trend caused primarily by human activity has many problems, not the least of which is that there has been an increase in global temperatures at the end of every interglacial period that had led (sometimes very quickly in a geological time frame) to new glacial advances and global cooling.
The science is not all in on this, and the science in no way dictates that the particular policies being bandied about by political bodies will have the desired effect. I am uncomfortable with the dogma that climate change must be linear and must be global warming, and that it must be stopped. And I worry that tying it to evolution--which is a demonstrated fact--is a mistake for the reasons I cite above.
In fact, I often wonder if the political global warming hysteria tends to demonstrate that most people do not in fact understand evolution, since adaptation to changes in environment is part of that process.
fnxtr · 25 July 2010
Uh-oh. I'm sure glad I'm not standing close to the fan...
Dale Husband · 25 July 2010
DS · 25 July 2010
eric · 25 July 2010
Clearly, MartinC, you have discovered a fatal flaw in this theory. Perhaps you'd like to tell us what alternate theory you have that explains the same phenomena?
And since your complaint seems to be that PZ's model allows for too wide a variety of different results, can we assume that your alternate theory has more stringent limitations on what things it predicts? After all, you would look pretty stupid if you first complained that the ToE is illegitimate because it allows for both fast and slow change, but then proposed an alternative that allows any change at any speed at all...right?
MrG · 25 July 2010
Matt Young · 25 July 2010
snaxalotl · 25 July 2010
the micro-macro blatherers are likely to think this post argues in their favor: we can get striking phenotype changes by winnowing alleles down to a smaller subset, hence useful evolution is accounted for by micro-evolution (winnowing) not macro-evolution (new alleles). perhaps it should be more readily conceded that (because of combination effects especially) changes in allele frequency generally produce a far more rapid response to natural selection than mutation - without making this explicit concession, creationists think they have a gotcha every time they think they see evidence that an initial diverse allele population can be filtered to produce every necessary species (despite the fact that these super allele populations somehow [megaploidy?] came off the ark). perhaps in making this concession more explicit, it will make more sense that functionally distinctive sub-populations provide very different environments (hence selection) for new mutations, regardless of whether the parent population has yet speciated. i don't want to tell people how to suck eggs, but i'm always seeing the micro-macro people unable to believe that useful new alleles are likely. the response to them is generally "mutation and selection is a fantastic mechanism for creating useful alleles", when it seems they need to hear "yes it does create useful alleles, but reconfiguration is fast and mutation is slow"
Oclarki · 25 July 2010
Sigh...I guess that I need a little help here.
Before asking for that help, though, a wee bit of biographical information. I am a geologist whose understanding of biology is limited to ecological risk assessments (particularly aquatic systems) and a rather insatiable desire to learn about stuff. My understanding of concepts like genetics, molecular biology, evo-devo and the like are improving (at least I think so) by virtue of certain popular-press books and by virtue of reading Nature cover-to-cover every week. Alas, that understanding is not even close to being sufficient to allow me to condense it down to simple compelling statements.
And simple, compelling statements are absolutely necessary to counter the claims of evolution deniers, especially the claims of deniers who merely parrot what they have been told. Lengthy and/ore overly technical explanations do not work so well...they tend to cause eyes to glaze over and brains to freeze up.
In the past I have tried to simplify the complex processes responsible for biological change as (primarily) natural selection (observed and also intuitive) working on variation (also observed and intuitive). Is this wrong? If so, how should I be defining evolution in a way that will not result in eyes-glazing and brain-freezing?
Scott F · 25 July 2010
As an educated lay person ("lay", relative to biology), I only recently came to appreciate the notion of evolution working on populations rather than on an individual lineage, mostly by reading here. I must admit that I was in the Bio 101 (or even Bio 10) camp. (And I thought I had been clever to understand the part where evolution operated on the "individual lineage", as opposed to operating on the individual.) The simple narrative made perfect sense, though there were some gaps in understanding, when moving up the complexity scale from the low level DNA mutations to the higher level "visible" changes in the species.
The hard part to grasp there is the time scale, and the mutability of the genome. That one "critical" mutation didn't happen just once in a single individual (in general, though it might). It happened many times in many individuals. I'm getting the impression (perhaps incorrectly) that the background level of mutation rates in a genome is kind of like the low level "quantum foam" of particle physics. (The analogy is far from perfect, but it's a helpful handle if I don't take it too seriously.) There are just lots and lots of "background" mutations happening all the time, and some times one of them "sticks" in the population, and becomes less random.
But as someone else noted, the more one learns about evolution, the more details that are discovered about how all the pieces fit together, the harder the whole picture is to refute. By all means, push as much of this detail as low in the curriculum as is practical. The details are comprehensible. It's that surface level understanding of evolution that is easier to refute. Get just a little past that surface level, and you'll have a convert for life.
(I know, I know, you've all gotten past that long before. I'm just trying to catch up. :-) Thanks for the continuing education.)
Steve P. · 25 July 2010
Mike Elzinga · 25 July 2010
Mike Elzinga · 26 July 2010
Steve P. · 26 July 2010
Mike Elzinga · 26 July 2010
Scott F · 26 July 2010
The "experts" in a scientific field may seem to the Creationist just like the "authority" figure in a religious context. But there's a difference. In science, anyone can become an expert, an "authority". All you have to do is study hard enough, learn, and earn a place at the table. You become an expert by being expert. In a religious context, "authority" isn't earned. It's either bestowed (by an existing Authority), or it's simply claimed or taken by fiat.
Similarly, I only provisionally accept the opinion of the science "expert". If I want to devote the time and energy, I can (in principle) prove to myself by myself whether the expert is right or not. In a religious context, there is simply no way, no matter how hard I study, or what experiments or observations I make, that I can confirm or deny the "revealed" word of God as given by the "authority" figure. If the revelation isn't given to me, it just isn't attainable even in principle. (In that sense, religion is very anti-democratic, very anti-capitalist.)
So, yes, I rely on the "authority" of those who know the science that I don't know. But only in so far as they prove to be authoritative.
Dale Husband · 26 July 2010
Mike Elzinga · 26 July 2010
SWT · 26 July 2010
Oclarki · 26 July 2010
Scott F · 26 July 2010
Oclarki · 26 July 2010
Mike Elzinga · 26 July 2010
Hygaboo Andersen · 26 July 2010
A look at the original article shows the author admits weak selection effects can not be measured. How convenient! Maybe that's because Darwinian selection itself doesn't exist and is an excuse for pornographers ans Sodomites to reject God!
Since the Darwiniacs who run that godless site want to charge mega-bucks for mere 24-hour access and demand that I register too, I would not survey the entire breadth of the excuse-making. The arrogance and greed of the Darwiniacs when it comes to their pathetic, deliberately abstruse "literature" is truly breathtaking!
Mike Elzinga · 26 July 2010
Man; you can sure see what topics stir up the lunatics.
Oclarki · 26 July 2010
Mike Elzinga · 26 July 2010
Oclarki · 26 July 2010
So....
One male gazelle is alert enough and quick enough to escape predation for at least one breeding cycle.
Another gazelle in the same herd is half a step slower and so becomes a belly-filler for lions, and consequently quite fails to breed.
How exactly does pornography and sodomy fit in to this?
Dale Lanz · 26 July 2010
Great article. I've wondered recently about how different gene combinations of already existing genetic material might be selectable, under changed enviromental conditions.
Robert Byers · 26 July 2010
This comment has been moved to The Bathroom Wall.
JGB · 26 July 2010
I appreciate the instinct to want to communicate quickly and efficiently to the public. Over the last couple of years of teaching though, I am somewhat skeptical of the value in emphasizing that as a strategy for educating. There are so many alternate conceptions that need to be properly changed (and the literature really hammers home how time consuming changing those conceptions is) that I think we are potentially kidding ourselves if we think a short 1-2 minute answer is going to have a lasting effect.
That said if your target is from an urban center transportation and road construction might be a useful metaphor for setting the stage to talk about mutation and network effects. The downside is that it leaves out an analogue to selection.
MrG · 26 July 2010
MrG · 26 July 2010
MrG · 26 July 2010
Vince · 26 July 2010
I teach my students that it's all about a Networks of Networks. The result is an emergent system with redundancy, resiliency, and "evolveability".
DS · 26 July 2010
MrG · 26 July 2010
Scott F · 26 July 2010
Tulse · 26 July 2010
DS · 26 July 2010
SWT · 26 July 2010
eric · 26 July 2010
Tulse · 26 July 2010
Mike Elzinga · 26 July 2010
I see that Ken Ham is blowing his usual gasket over evolution by carping about the National Museum of Natural History.
This is where our trolls get their understanding of science; from a charlatan fomenting the culture wars while building an empire bamboozling fundamentalists and selling home schooling materials to the very people he fills with paranoia and hatred.
JGB · 26 July 2010
I wonder if Ham or Todd Wood has properly read Louis Agassiz. I don't think he would have approved of their heretical notion of kinds and baramins (aka we will refute evolution as true by assuming it actually happens). He was so dedicated to the notion of types that he very much doubted that dogs only had one common ancestor. I just read the Dover reprint of his Essay on Classification. Some interesting nuggets from the last actual attempt of an actually scientific creationism. Overall not that exciting, except for its account of another mutually contradictory creationist theory.
SWT · 26 July 2010
Tulse · 26 July 2010
Ray Martinez · 26 July 2010
The main complaint of Myers is that evolution is being overly simplified---to the point of error.
He makes this point in reference to evolutionist and creationist alike.
Then Myers launches into a very long and complicated explanation of how evolution proceeds. This is why evolution is overly simplified in the first place.
MrG · 26 July 2010
Shebardigan · 26 July 2010
henry · 27 July 2010
harold · 27 July 2010
Dale Husband · 27 July 2010
harold · 27 July 2010
Henry -
I believe in some government funding for scientific education of the general public, but at least we agree on the more important priority, that creationist museums shouldn't be publicly funded.
As for the contention that there are no mainstream science museums that receive mainly private funding, http://en.wikipedia.org/wiki/American_Museum_of_Natural_History
Frank J · 27 July 2010
harold · 27 July 2010
Frank J -
At this point, either "ID" or "creationism" borders on being a euphemism.
Operationally, it's now just evolution denial.
All positive claims have been stripped away and all the up to date adherents know better than to make any testable claim that any deity did any specific thing at any time. There is actually virtually no reference to any act of "creation" or "intelligent design" whatsoever - just a lot of dishonest arguments against "Darwinism".
In theory, some sort of evolution denial might exist that they might be leery of including in their tent, but in practice, I'm not aware of any.
"Say anything to deny the theory of evolution and agree with anyone who says anything to deny the theory of evolution" is the current observable behavior.
Now, if I were to conjecture as to why this behavior exists, I would, as you already know, conjecture that, in the post-civil rights milieu, Biblical literalism was recruited as a "moral" justification for harsh political ideals, but that at the same time, biomedical science was exploding, leading to the frenzied "creation science" of the sixties and seventies - a panicky reaction to the dawn of molecular genetics, among other things (which drew on older, mainly theology-inspired traditions). However, "creation science" lost in court, leading to a reflexive emotional obsession with "denying evolution" by any means possible, largely as a way of denying the defeat. As I've noted before, the contemporary language of evolution denial is all about obfuscation and plausible deniability.
But while conjecture is conjecture, observable behavior is observable.
Ray Martinez · 27 July 2010
Frank J · 27 July 2010
Oclarki · 27 July 2010
MrG · 27 July 2010
Oh, now you done it ... you fed da troll.
Oclarki · 27 July 2010
henry · 28 July 2010
Dave Luckett · 28 July 2010
For the same reason that there is funding for science, ignoramus. The theory of evolution is science. Science is what drives technology, and the public benefits from it. Creationism, on the other hand, is a regression to the dark ages, and the public would suffer from it.
Dale Husband · 28 July 2010
Stanton · 28 July 2010
DS · 28 July 2010
MrG · 28 July 2010
Kevin · 28 July 2010
Sorry, I'm a bit late to the party here, but have a question for any experts still reading the thread. Would this indicate that if Dr. Lenski were to blend 4 or 5 of his different lines of non-Citrate-metabolizing bacteria, thus creating a population with a greatly increased allelic variety, then he would expect the ability to metabolize citrate to evolve faster than it was able to otherwise? Or does the asexual reproductive nature of e. coli preclude the effect?
Ray Martinez · 28 July 2010
Ray Martinez · 28 July 2010
MrG · 28 July 2010
Oh gosh, it's evening, feeding time for the troll.
Oclarki · 28 July 2010
MrG · 28 July 2010
Ray Martinez · 28 July 2010
MrG · 28 July 2010
On googling the name, add "creationist" to the search term -- it's a common name.
Oclarki · 28 July 2010
Oclarki · 28 July 2010
MrG · 28 July 2010
tresmal · 28 July 2010
Oclarki · 28 July 2010
Dale Husband · 29 July 2010